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I study how ion channel dynamics and glial-axonal metabolic cross-talk drive neurodegeneration. This focus grew from my early work using electrophysiology to investigate sodium channel dysfunction in epileptic encephalopathies. I later transitioned to studying glial dysfunction in CMT1X neuropathy, examining how Connexin-32 mutations compromise Schwann cell and axon health. Currently, I explore the metabolic crosstalk between Schwann cells and axons, investigating how glial-derived metabolite exchange buffers against SARM1-mediated axonal degeneration to ensure long-term neural survival.
